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| Early in mammalian development, all but one of the X chromosomes in each cell of an embryonic female is inactivated. X-chromosome inactivation (XCI) is one of the earliest events in developmental gene regulation, and provides an important model for gene regulation in general. Errors in gene regulation are responsible for many genetic disorders and for some changes that characterize cancerous cells. Further, abnormalities of the X chromosome occur frequently in humans, and sporadic reactivation of all or part of the inactive X chromosome is one of the few gene regulatory changes clearly associated with aging.
The DNA of a single chromatin domain shares a particular level of sensitivity to enzymes that cleave DNA (nucleases). Such nuclease sensitivity can be readily assayed by techniques we have already used on a much smaller scale to characterize the genes themselves. We plan to define length of DNA at a particular level of nuclease sensitivity for the Xist gene, in order to map the probable extent of the domain. These experiments involve isolation of nuclei from cultured cells or from fresh liver cells, treatment of these nuclei with controlled concentrations of the nuclease, DNase I, gel fractionation of the resulting DNA fragments, and characterization of those fragments using cloned molecular probes.
Last modified July 10, 2002 |
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